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00:22
1.
index 1
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00:21
2.
Gastric cancer (GC)
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00:22
3.
Gastric cancer (GC)
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00:14
4.
Human Genome
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00:22
5.
Circular RNA (CircRNA)
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00:30
6.
Biogenesis of circRNA
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00:35
7.
CircRNA circularization - Alu element
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00:27
8.
CircRNA circularization – RNA Binding Protein (RBP)
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00:33
9.
Functions of circRNA
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00:20
10.
The role of circRNAs in cancer
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00:58
11.
Results
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00:38
12.
Results
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00:16
13.
Aim
-
00:32
14.
Results
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00:47
15.
CircPDIA4 promoted migration and metastasis capabilities of gastric cancer cells in vitro and in vivo
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00:56
16.
CircPDIA4 promoted migration and metastasis capabilities of gastric cancer cells in vitro and in vivo
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00:15
17.
Summary
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00:07
18.
Slide 18
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00:42
19.
The RBP QKI induced the formation of circPDIA4 in gastric cancer cells
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00:20
20.
The RBP QKI induced the formation of circPDIA4 in gastric cancer cells
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00:23
21.
The RBP QKI induced the formation of circPDIA4 in gastric cancer cells
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00:29
22.
The RBP QKI induced the formation of circPDIA4 in gastric cancer cells
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00:23
23.
Summary
-
00:05
24.
Slide 24
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01:14
25.
CircPDIA4 bound with DHX9 and ERK1/2
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01:08
26.
CircPDIA4 sustained high phosphorylation levels of ERK1/2
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00:59
27.
CircPDIA4 sustained high phosphorylation levels of ERK1/2 through interrupting binding of DUSP6 with ERK1/2
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00:29
28.
CircPDIA4 sustained high phosphorylation levels of ERK1/2 through interrupting binding of DUSP6 with ERK1/2
-
00:31
29.
CircPDIA4 sustained high phosphorylation levels of ERK1/2 and reduced the sensitivity of ERKi
-
00:00
30.
Summary
-
00:01
31.
CircPDIA4 competitively combined with DHX9 as a decoy to promote oncogenic circRNAs formation
-
00:22
32.
Summary
-
01:03
33.
CircPDIA4 competitively combined with DHX9 as a decoy to promote oncogenic circRNAs formation
-
00:41
34.
CircPDIA4 competitively combined with DHX9 as a decoy to promote oncogenic circRNAs formation
-
00:46
35.
CircPDIA4 competitively combined with DHX9 as a decoy to promote oncogenic circRNAs formation
-
00:35
36.
CircPDIA4 competitively combined with DHX9 as a decoy to promote oncogenic circRNAs formation
-
00:26
37.
Summary
-
00:19
38.
Conclusion
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00:14
39.
Conclusion
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00:15
40.
Conclusion
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00:24
41.
Pros :
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00:02
42.
Slide 40
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00:01
43.
Slide 41
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26:11
44.
** after 20230307 seminar - 複本.pptx