• 00:00 1.
    index 1
  • 00:02 2.
    Slide 1
  • 00:40 3.
    Cigarette smoking promotes cell proliferation and malignancy of NSCLC cells
  • 01:45 4.
    ** after 20240128 lab meeting.pptx
  • 01:50 5.
    Slide 18
  • 01:52 6.
    Slide 19
  • 01:53 7.
    Slide 20
  • 01:56 8.
    Slide 19
  • 01:56 9.
    Slide 20
  • 01:57 10.
    ** after 20240128 lab meeting.pptx
  • 02:22 11.
    Slide 18
  • 02:22 12.
    Slide 0
  • 02:22 13.
    Slide 18
  • 02:54 14.
    ** after 20240128 lab meeting.pptx
  • 03:00 15.
    Slide 18
  • 03:16 16.
    ** after 20240128 lab meeting.pptx
  • 05:32 17.
    Cigarette smoking promotes cell proliferation and malignancy of NSCLC cells
  • 06:00 18.
    Cigarette smoking promotes cell proliferation and malignancy of NSCLC cells
  • 13:11 19.
    The m6A modification of CDK2AP2 mRNA is involved in a CSE-induced cell cycle disorder in NSCLC cells
  • 23:00 20.
    The m6A modification of CDK2AP2 mRNA is involved in a CSE-induced cell cycle disorder in NSCLC cells
  • 40:12 21.
    Upon CSE treatment, HIF-1α activates METTL3 transcription in NSCLC cells
  • 40:14 22.
    METTL3 is involved in the CSE-induced m6A modification, cell proliferation, and malignancy of NSCLC cells
  • 40:14 23.
    METTL3 is involved in the CSE-induced m6A modification, cell proliferation, and malignancy of NSCLC cells
  • 40:15 24.
    HIF-1α is involved in the CSE-induced m6A modification, cell proliferation, and malignancy of NSCLC cells
  • 40:16 25.
    METTL3 is involved in the CSE-induced m6A modification, cell proliferation, and malignancy of NSCLC cells
  • 40:16 26.
    METTL3 is involved in the CSE-induced m6A modification, cell proliferation, and malignancy of NSCLC cells
  • 40:17 27.
    Upon CSE treatment, HIF-1α activates METTL3 transcription in NSCLC cells
  • 40:18 28.
    The m6A modification of CDK2AP2 mRNA is involved in a CSE-induced cell cycle disorder in NSCLC cells
  • 40:18 29.
    The m6A modification of CDK2AP2 mRNA is involved in a CSE-induced cell cycle disorder in NSCLC cells
  • 40:22 30.
    The m6A modification of CDK2AP2 mRNA is involved in a CSE-induced cell cycle disorder in NSCLC cells
  • 41:19 31.
    Upon CSE treatment, HIF-1α activates METTL3 transcription in NSCLC cells
  • 1:02:35 32.
    METTL3 is involved in the CSE-induced m6A modification, cell proliferation, and malignancy of NSCLC cells
  • 1:06:50 33.
    METTL3 is involved in the CSE-induced m6A modification, cell proliferation, and malignancy of NSCLC cells
  • 1:09:01 34.
    HIF-1α is involved in the CSE-induced m6A modification, cell proliferation, and malignancy of NSCLC cells
  • 1:09:02 35.
    METTL3 is involved in the CSE-induced m6A modification, cell proliferation, and malignancy of NSCLC cells
  • 1:09:09 36.
    HIF-1α is involved in the CSE-induced m6A modification, cell proliferation, and malignancy of NSCLC cells
  • 1:09:14 37.
    HIF-1α is involved in the CSE-induced m6A modification, cell proliferation, and malignancy of NSCLC cells
  • 1:09:15 38.
    HIF-1α, via METTL3 regulation of the CDK2AP2 m6A modification, is involved in CSE-induced cell proliferation and malignancy of NSCLC cells
  • 1:12:19 39.
    HIF-1α, via METTL3 regulation of the CDK2AP2 m6A modification, is involved in CSE-induced cell proliferation and malignancy of NSCLC cells
  • 1:12:19 40.
    HIF-1α, via METTL3 regulation of the CDK2AP2 m6A modification, is involved in promoting the neoplastic capacity of CSE-NSCLC cells in xenografts of nude mice
  • 1:12:26 41.
    HIF-1α, via METTL3 regulation of the CDK2AP2 m6A modification, is involved in CSE-induced cell proliferation and malignancy of NSCLC cells
  • 1:12:57 42.
    HIF-1α, via METTL3 regulation of the CDK2AP2 m6A modification, is involved in promoting the neoplastic capacity of CSE-NSCLC cells in xenografts of nude mice
  • 1:15:25 43.
    HIF-1α, via METTL3 regulation of the CDK2AP2 m6A modification, is involved in promoting the neoplastic capacity of CSE-NSCLC cells in xenografts of nude mice
  • 1:15:55 44.
    For smokers with NSCLC, the levels of HIF-1α and METTL3 are elevated and those of CDK2AP2 are lowered
  • 1:16:52 45.
    Slide 16
  • 1:16:55 46.
    For smokers with NSCLC, the levels of HIF-1α and METTL3 are elevated and those of CDK2AP2 are lowered
  • 1:18:41 47.
    Slide 16
  • 1:19:27 48.
    ** after 20240128 lab meeting.pptx
  • 索引
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  • 全螢幕
20240129 lab meeting_照穎
長度: 1:19:40, 瀏覽: 86, 最近修訂: 2024-01-29
    上一篇 下一篇
    • 00:00 1.
      index 1
    • 00:02 2.
      Slide 1
    • 00:40 3.
      Cigarette smoking promotes cell proliferation and malignancy of NSCLC cells
    • 01:45 4.
      ** after 20240128 lab meeting.pptx
    • 01:50 5.
      Slide 18
    • 01:52 6.
      Slide 19
    • 01:53 7.
      Slide 20
    • 01:56 8.
      Slide 19
    • 01:56 9.
      Slide 20
    • 01:57 10.
      ** after 20240128 lab meeting.pptx
    • 02:22 11.
      Slide 18
    • 02:22 12.
      Slide 0
    • 02:22 13.
      Slide 18
    • 02:54 14.
      ** after 20240128 lab meeting.pptx
    • 03:00 15.
      Slide 18
    • 03:16 16.
      ** after 20240128 lab meeting.pptx
    • 05:32 17.
      Cigarette smoking promotes cell proliferation and malignancy of NSCLC cells
    • 06:00 18.
      Cigarette smoking promotes cell proliferation and malignancy of NSCLC cells
    • 13:11 19.
      The m6A modification of CDK2AP2 mRNA is involved in a CSE-induced cell cycle disorder in NSCLC cells
    • 23:00 20.
      The m6A modification of CDK2AP2 mRNA is involved in a CSE-induced cell cycle disorder in NSCLC cells
    • 40:12 21.
      Upon CSE treatment, HIF-1α activates METTL3 transcription in NSCLC cells
    • 40:14 22.
      METTL3 is involved in the CSE-induced m6A modification, cell proliferation, and malignancy of NSCLC cells
    • 40:14 23.
      METTL3 is involved in the CSE-induced m6A modification, cell proliferation, and malignancy of NSCLC cells
    • 40:15 24.
      HIF-1α is involved in the CSE-induced m6A modification, cell proliferation, and malignancy of NSCLC cells
    • 40:16 25.
      METTL3 is involved in the CSE-induced m6A modification, cell proliferation, and malignancy of NSCLC cells
    • 40:16 26.
      METTL3 is involved in the CSE-induced m6A modification, cell proliferation, and malignancy of NSCLC cells
    • 40:17 27.
      Upon CSE treatment, HIF-1α activates METTL3 transcription in NSCLC cells
    • 40:18 28.
      The m6A modification of CDK2AP2 mRNA is involved in a CSE-induced cell cycle disorder in NSCLC cells
    • 40:18 29.
      The m6A modification of CDK2AP2 mRNA is involved in a CSE-induced cell cycle disorder in NSCLC cells
    • 40:22 30.
      The m6A modification of CDK2AP2 mRNA is involved in a CSE-induced cell cycle disorder in NSCLC cells
    • 41:19 31.
      Upon CSE treatment, HIF-1α activates METTL3 transcription in NSCLC cells
    • 1:02:35 32.
      METTL3 is involved in the CSE-induced m6A modification, cell proliferation, and malignancy of NSCLC cells
    • 1:06:50 33.
      METTL3 is involved in the CSE-induced m6A modification, cell proliferation, and malignancy of NSCLC cells
    • 1:09:01 34.
      HIF-1α is involved in the CSE-induced m6A modification, cell proliferation, and malignancy of NSCLC cells
    • 1:09:02 35.
      METTL3 is involved in the CSE-induced m6A modification, cell proliferation, and malignancy of NSCLC cells
    • 1:09:09 36.
      HIF-1α is involved in the CSE-induced m6A modification, cell proliferation, and malignancy of NSCLC cells
    • 1:09:14 37.
      HIF-1α is involved in the CSE-induced m6A modification, cell proliferation, and malignancy of NSCLC cells
    • 1:09:15 38.
      HIF-1α, via METTL3 regulation of the CDK2AP2 m6A modification, is involved in CSE-induced cell proliferation and malignancy of NSCLC cells
    • 1:12:19 39.
      HIF-1α, via METTL3 regulation of the CDK2AP2 m6A modification, is involved in CSE-induced cell proliferation and malignancy of NSCLC cells
    • 1:12:19 40.
      HIF-1α, via METTL3 regulation of the CDK2AP2 m6A modification, is involved in promoting the neoplastic capacity of CSE-NSCLC cells in xenografts of nude mice
    • 1:12:26 41.
      HIF-1α, via METTL3 regulation of the CDK2AP2 m6A modification, is involved in CSE-induced cell proliferation and malignancy of NSCLC cells
    • 1:12:57 42.
      HIF-1α, via METTL3 regulation of the CDK2AP2 m6A modification, is involved in promoting the neoplastic capacity of CSE-NSCLC cells in xenografts of nude mice
    • 1:15:25 43.
      HIF-1α, via METTL3 regulation of the CDK2AP2 m6A modification, is involved in promoting the neoplastic capacity of CSE-NSCLC cells in xenografts of nude mice
    • 1:15:55 44.
      For smokers with NSCLC, the levels of HIF-1α and METTL3 are elevated and those of CDK2AP2 are lowered
    • 1:16:52 45.
      Slide 16
    • 1:16:55 46.
      For smokers with NSCLC, the levels of HIF-1α and METTL3 are elevated and those of CDK2AP2 are lowered
    • 1:18:41 47.
      Slide 16
    • 1:19:27 48.
      ** after 20240128 lab meeting.pptx
    位置
    1. 知識庫
    2. ...
    3. 2024
    資料夾名稱
    2024
    發表人
    周楷軒
    單位
    賴亮全教授
    建立
    2024-01-29 10:38:40
    最近修訂
    2024-01-29 11:03:26
    長度
    1:19:40

    臺北市仁愛路一段1號10樓1009室

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    E-mail: llai@ntu.edu.tw

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    Lab: 886-2-2393-8235